Question: Hello and thank you for your time and input.

I've had these symptoms intermittently for the past year or so.
`Heaviness/painful/hot sensation in legs when in hot temperature/hot shower. This goes away quickly once out of heat.
`Cognitive difficulties/concentration/working memory issues. Neuropsychology test showed a decline in certain functioning areas as compared to a previous test from two years prior.
`Walking up to the third floor, HR averages 131 with shortness of breath.
`On-going fatigue
`Sudden pain down legs which causes leg to 'give out' and cause a fall from loss of support.
`Very irregular menses without clear cause.
`Occasional unfocused vision.
`Occasional trembling of hands/fingers.
`Dizziness/blurred/blackness in vision when going from sitting or squatting to standing.
`MRI of brain showed some scattered areas of demyelination.
`Spinal tap- all normal
`Negative lupus test

Doctor suspects MS but currently unsure.

I did read that CSF is normal in approximately 20% of MS patients.
I am HIV negative, tested recently.
What is 'evoked potential'?

MRI information:


**********
BRAIN MRI RESULTS:

Exam Information
Modality: MR
Body Part: BRAIN
Description: MRI BRAIN W & W/O XXXXXXX
Performed Date: 5/19/2016 6:56:04
Patient History: Reason for Study: R/O MULTIPLE SCLEROSIS
REQUESTED EXAM: MRI BRAIN REASON FOR EXAM:
Final Report
PROCEDURE:
967^MRI BRAIN W & W/O DYE^L
IMPRESSION:

A few scattered nonspecific areas of increased T2 deep white matter signal,
including one which appears to enhance raising the suspicion for demyelinating
disease. Differential diagnosis, however, is broad and includes infectious and
noninfectious etiologies.

4^ABNORMALITY, ATTN. NEEDED^L
REPORT:
MR examination of the brain with contrast.

Clinical indication: Multiple sclerosis

Comparison: None

Description:

Axial T1, T2, FLAIR, diffusion, sagittal T1, and coronal T1 weighted images
obtained prior to the administration intravenous gadolinium. Following the
administration of intravenous gadolinium, sagittal, axial and coronal
T1-weighted images were obtained.

There is no acute infarct. There is no intracranial hemorrhage. There are no
subdural or epidural collections. The pituitary gland measures 1.1 x 1.2 x 0.6
cm and appears convex. There are a few scattered nonspecific areas of increased
T2 deep white matter signal, which are nonspecific but not anticipated in a
patient in this age category. Differential diagnosis is broad but includes
demyelinating disease. One of these lesions in the right preinsula region
demonstrates contrast enhancement raising the suspicion for demyelinating
disease. There are no masses. The ventricles appear midline. Normal vascular
flow voids are present. The visualized posterior fossa is unremarkable. The
paranasal sinuses are clear.



**********************
CERVICAL SPINE MRI RESULTS

Body Part: CERVICAL S
Description: MRI CERVICAL SPINE W/O & W CONTRAST
Performed Date: 5/19/2016 6:56:04
Patient History: Reason for Study: R/O MULTIPLE SCLEROSIS Patient's REQUESTED EXAM: MRI C-SPINE REASON FOR EXAM
Final Report
PROCEDURE:
973^MRI CERVICAL SPINE W/O & W CONTRAST^L
IMPRESSION:
1. Mild degenerative disc changes. 2. Normal-appearing cervical spine cord
with no suspicious lesion seen within the cervical spinal cord. 3. Most likely
flow artifact in the upper thoracic spinal canal on the sagittal STIR and
T2-weighted series only.

2^MINOR ABNORMALITY^L
REPORT:
Technique: Standard technique for pre and post gadolinium enhanced cervical
spine MRI performed.

Report: The spinal cord appears normal in shape and signal with no abnormally
hyperintense lesions or enhancing lesion seen. Along the posterior margin of
the upper thoracic spinal cord, within the spinal canal, there are ill-defined
T2-weighted hypointense signal changes with no corresponding areas of signal
abnormality seen on the T1 weighted series or on the post gadolinium series or
on the T2-weighted axial images..

Mild degenerative changes are noted throughout the mid and lower cervical
spine. The most severe changes are present at the C5-6 interspace where there
is mild to moderately advanced posterior disc osteophyte complex but no spinal
canal stenosis, discrete disc protrusion or severe neural foraminal narrowing.


**********************
LUMBER MRI RESULTS

Body Part: LUMBAR SPI
Description: MRI LUMBAR W/O CONTRAST
Performed Date: 5/19/2016 6:56:04
Patient History: Reason for Study: R/O MULTIPLE SCLEROSIS
REQUESTED EXAM: LOW BACK PAIN AND DYSESTHESIA
Final Report
PROCEDURE:
977^MRI LUMBAR W/O CONTRAST^L
IMPRESSION:
Moderately advanced degenerative changes at levels as noted above along with a
moderate-sized disc protrusion at L5-S1.

2^MINOR ABNORMALITY^L
REPORT:
Technique: Standard technique for non gadolinium-enhanced lumbar spine MRI
performed.

Report: Moderately severe degenerative disc changes are noted throughout the
lumbar spine. A 10 mm hemangiomatous appearing lesion is present in the L1
vertebral body. The conus tapers normally at the level of L1. Visualized
portions of the spinal cord appear normal.

Level specific findings are as follows:

L1-2: Small left paracentral to lateral protrusion. No spinal canal stenosis.
No severe neural foraminal narrowing.

L2-3: No severe degenerative changes

L3-4: Mild to moderate broad-based posterior disc bulging along with mild
degenerative changes of the posterior elements. There is mild narrowing of the
spinal canal but not stenotic. No discrete disc protrusion present. Mild
bilateral neural foraminal narrowing present.

L4-5: Somewhat more prominent degenerative changes involving the posterior
elements and disc at this level compared to L3-4. Mild to moderately severe
bilateral neural foraminal narrowing present. No stenosis or discrete disc
protrusion.

L5-S1: Moderate-sized central disc protrusion with disc material extending up
to the ventral margin of both descending S1 nerve roots. The nerve roots
however do not appear displaced and there does appear to be at least a with
spurring of fluid signal present between the S1 nerves and the disc protrusion.
Mild bilateral neural foraminal narrowing.