What causes hunt syndrome?
User rating for this question
XXXXXXX hunt sndrome
Posted Thu, 20 Feb 2014 in Brain and Spine
Answered by Dr. Suleman Muhammad 1 hour later
Brief Answer: RHS a self resolving VZV infection Detailed Answer: Good Day, I am Dr Suleman and I would like to help you know what XXXXXXX hunt syndrome is. At the outset, XXXXXXX hunt syndrome is termed for more than 1 condition. Of those the most common XXXXXXX Hunt syndrome is a neuropathy of facial area with associated ear canal and mucous membrane involvement also called Herpes Zoster Oticus. It was first described in 1907 by Dr XXXXXXX XXXXXXX on a patient who had ear pain with associated rashes on skin and mucosa by human herpes virus 3 infecting geniculate ganglion.Classic XXXXXXX Hunt syndrome is ascribed to infection of the geniculate ganglion by herpesvirus 3 (varicella-zoster virus [VZV]). It usually occurs in people who had chicken pox in the past and the pox virus VZV remain dormant in the neurons and later show up as XXXXXXX hunt. But is not always correlated with chicken pox. XXXXXXX Hunt is not lethal but rather self resolving only trouble is facial weakness and recovery is less than 50 % of facial palsy. XXXXXXX Hunt syndrome has following presentation Vesicular rash of the ear or mouth (as many as 80% of cases) The rash might precede the onset of facial paresis/palsy (involvement of the seventh cranial nerve [CN VII]) Ipsilateral lower motor neuron facial paresis/palsy (CN VII) Vertigo and ipsilateral hearing loss (CN VII) Tinnitus (buzzing ear sound) Otalgia (Ear Pain) Headaches Dysarthria( Difficulty speaking) Gait ataxia (Gait disturbance) Fever Cervical lymph nodes involvement Facial weakness usually reaches maximum severity by 1 week after the onset of symptoms. Other cranial neuropathies might be present and may involve cranial nerves (CNs) VIII, IX, X, V, and VI. Ipsilateral hearing loss has been reported in as many as 50% of cases. Blisters of the skin of the ear canal, auricle, or both may become secondarily infected, causing cellulitis. Poor prognostic factors for good functional recovery include the following: Age older than 50 years Complete facial paralysis Lack of CN VII nerve excitability Corticosteroids and oral acyclovir are commonly used in the treatment of XXXXXXX Hunt syndrome.Vestibular suppressants ,Anticholinergics, antihistamines, opioids and local anesthestics have also proved useful. A team of otorhinolaryngologist, opthalmologist, infectious disease specialist and neurosurgeons is required to manage the RHS. Hope you got what you were looking for XXXX Take care