LDL is a low density lipoprotein. Lipoproteins
are what carry lipids
around our body. An LDL starts life as a Very Low Density Lipoprotein (VLDL), loaded with fats in the form of trigycerides and cholesterol. As it gives out the tryglycerides to cells for storage or energy, it eventually becomes an LDL containing mainly cholesterol. Now, every cell in our body needs cholesterol, that is why the liver produces it, so when a cell needs cholesterol it puts out a receptor so the LDL can dock into the cell. The LDL is then, in effect, swallowed up by the cell, dismantled and the cholesterol becomes available for use.
If all the cells in the body have enough cholesterol then they will not have the receptor, so the LDL cannot be utilised.
An LDL is no more sticky than any other lipoprtein, regardless of the garbage that others want to spew forth.
They can though be found in the inflammation surrounding an injury
. They don't cause the injury (unless they are oxidised) just are part of the body's response. When something damages the lining of the artery
, inflammation occurs and LDLs will be there in the body's attempt to utilise the cholesterol as part of the repair process.
Some of the people who suffer a heart attack
will have elevated levels of LDL. Whatever causes this, is most likely what causes the heart attack
Nothing simply adheres to walls of out arteries (I can't beleive a nurse can be so ignorant), they are designed for this not to happen. Something must first damage the "non stick" lining called the endothelium