on going UTI (or so the dr. thought) Had a catscan and it revealed a thickening of the bladder. Bacteria and blood in urine sample. What causes "thickening" of the bladder?

what is a cysto test and how does it go?

i have what seams like air comeing from my penice when i urinate can you help whit this problem

Hi,

I just had a ultrasound on my kidneys and bladder and the results were "Unremarkable Renal Ultrasound" Am I correct that this means normal?

Also my bladder was "underdistended" what does that mean?

Thanks for your help!

What conditions or diseases are associated with blood in the urine, and if needed, what specialty doctor do I need to see?

Intermittant lower abdominal cramping & fatique. No constipation.

Due to mild abdominal pain and recent weight loss, I was sent for a CT session of "Pre-contrast Helical scans of the abdomen and pelvis + Post-contrast Axial scans of the abdomen and pelvis during the venous phase and of the urinary bladder after 7-mins-delay" - A question here: what is the purpose of a 7-mins-delsy scan of the urinary bladder?? I did not say I feel anything wrong with my urinary/bladder function..
Thank you Doctor for your advice.

i Had a pet/ct.
can you review and give your view
TUMOR IMAGE PET/CT SKUL-THIGH (CPT=78815) - Details
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About This Test

Details
Study Result

WHOLE BODY PET-CT, 2/26/2015.

COMPARISON STUDIES:
CT of the abdomen and pelvis, 5/9/2014.

CLINICAL HISTORY:
Transitional cell adenocarcinoma of the urinary bladder, stage II..
Cystitis cystica.

IMAGING PROTOCOL:
Whole body PET-CT images were obtained, scanning from the skull base to the proximal femurs, 60
minutes following the uneventful intravenous administration of 12.6 mCi of Flouorine-18 FDG,
including a low-dose noncontrast CT scan, for localization-fusion purposes, followed by 2-D axial
PET images, supplemented by reformatted coronal and sagittal fused PET-CT images.
Automated exposure control and ALARA manual techniques for patient specific dose reduction were
followed while maintaining the necessary diagnostic image quality.

ADVERSE EFFECTS:
None.

FINDINGS:
THE NECK:
The nasopharynx, oropharynx, and hypopharynx maintain grossly normal nonenhanced CT appearances,
without definite discrete hypermetabolic lesions.
The parotid and submandibular glands are normal and symmetrical in size, without discrete
hypermetabolic lesions.
The larynx has a normal CT appearance, without definite focal nonenhanced CT abnormalities or
discrete hypermetabolic lesions.

No pathologically enlarged or hypermetabolic cervical or supraclavicular lymph nodes are present.

The thyroid gland is normal in size, without definite dominant solid nodules or discrete
hypermetabolic lesions.

THE THORAX:
No suspicious hypermetabolic pulmonary nodules are detected.
The lungs are normal in volume, without airspace consolidation or pleural effusions.

The thoracic aorta and central pulmonary arteries are normal in caliber; the heart chambers are
normal in size, without pathological hypermetabolic pericardial radiotracer uptake or a pathological
pericardial effusion; multivessel calcified coronary plaque is present.

No pathologically enlarged or hypermetabolic axillary, mediastinal, hilar, or cardiophrenic lymph
nodes are present.

THE ABDOMEN:
The liver is normal in size and morphology, demonstrating homogeneous parenchymal attenuation and
normal heterogeneous physiological tracer uptake, without definite discrete focal parenchymal CT
abnormalities or pathological hypermetabolic foci that exceed a 2:1 ratio, in comparison to the
normal hepatic parenchyma.
The pancreas is normal in size and morphology, demonstrating homogeneous glandular attenuation,
without definite discrete focal CT abnormalities or pathological hypermetabolic foci.
The spleen is normal in size and attenuation, without pathological hypermetabolic foci.
Stable bilateral adrenomegaly is redemonstrated, consistent with adrenal hypertrophy, without
dominant nodules or pathological hypermetabolic foci.
The kidneys are normal and symmetrical in size and cortical thickness, without pyelocaliectasis or
perinephric stranding; both kidneys demonstrate homogeneous nephrographic attenuation, without
definite discrete solid lesions.

Diffuse aortoiliac atherosclerosis is present, without aneurysmal dilatation.

No pathologically enlarged or hypermetabolic retroperitoneal lymph nodes are present.

THE PELVIS:
The urinary bladder is partially distended, demonstrating mild-moderate circumferential wall
thickening, accompanied by intense edematous perivesical fat stranding, without discrete
intraluminal filling defects; a small diverticulum is present along the left lateral bladder wall.
Mild prostatomegaly is present, associated with multiple similar foci of intense hypermetabolic
radiotracer uptake along the peripheral zone.

No pathologically enlarged or hypermetabolic pelvic or inguinal lymph nodes are present.

The small and large bowel are normal in caliber and wall thickness, demonstrating extensive sigmoid
diverticular formation, without discrete hypermetabolic intraluminal lesions or edematous
mesenteric-pericolic fat stranding.

No pelvic ascites is present.

THE SKELETAL SYSTEM:
The cervicothoracolumbar vertebra maintain normal heights, without discrete blastic or lytic
vertebral lesions or discrete hypermetabolic foci.
No discrete hypermetabolic osseous lesions are detected along the thoracic cage, the proximal
humeri, or the proximal femurs.

=====
IMPRESSION:
1. Mild-moderate circumferential wall thickening of the urinary bladder wall, accompanied by
intense edematous perivesical fat stranding, collectively reflecting the clinical history of
cystitis cystica, without discrete intraluminal filling defects.
2. Multiple similar foci of intense hypermetabolic radiotracer uptake along the prostatic
peripheral zone, highly suspicious for multifocal prostatic carcinoma; the differential diagnosis
does include multifocal prostatitis.
3. No PET/CT evidence of remote hepatic, pulmonary, or osseous metastases.
4. No PET/CT evidence of hypermetabolic pelvic nodal disease.
5. Coronary atherosclerosis.
6. Diffuse aortoiliac atherosclerosis, without aneurysmal dilatation.
7. Extensive sigmoid diverticular formation.

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mild pelvicalyceal dilatation means? what is the problem? if it is problem how we can solve this?